top of page

PTSD and C-PTSD: Two Different Psychological Experiences

  • Mar 6
  • 7 min read

Article written in collaboration with @ppsycotips


When trauma leaves different traces

Psychological trauma is one of the most complex and devastating human experiences. Yet not all traumas resemble one another, and not all of their consequences follow the same path. In recent decades, clinical research has clarified a fundamental distinction: that between Post-Traumatic Stress Disorder (PTSD) and Complex Post-Traumatic Stress Disorder (C-PTSD). Understanding this difference is not merely an academic exercise — it is an act of care toward people who carry the weight of prolonged traumatic experiences.


PTSD: when a single event shatters equilibrium

PTSD was officially recognized as a diagnostic category in 1980, in the DSM-III, largely thanks to studies on Vietnam War veterans. Since then its definition has been refined, but the core remains unchanged: PTSD develops following a single or time-limited traumatic event — a serious accident, an assault, a natural disaster, a wartime experience — that exceeds the individual's capacity to process and integrate the experience.


PTSD symptoms are organized around four main clusters: intrusion (flashbacks, nightmares, intrusive memories), avoidance (of trauma-associated stimuli), negative alterations in cognition and mood, and hyperactivation of the autonomic nervous system (American Psychiatric Association [APA], 2022). The common denominator is that the brain continues to react as if the danger were still present, unable to "file away" the event in the past.


From a neurobiological standpoint, PTSD is associated with chronic hyperactivation of the amygdala — the brain structure responsible for threat processing — and reduced activity of the prefrontal cortex, which normally exercises inhibitory control over alarm responses (van der Kolk, 2014). In parallel, a reduction in hippocampal volume is frequently observed, a structure crucial for the spatio-temporal contextualization of memories: this explains why flashbacks are not experienced as memories of the past, but as events happening in the present (Bremner, 2006).


C-PTSD: when trauma inhabits relationships

The concept of C-PTSD emerged from the pioneering work of Judith Herman, who in 1992 observed that survivors of prolonged and repeated trauma — particularly women who were victims of domestic violence and individuals with histories of childhood abuse — presented a constellation of symptoms that the classical PTSD model failed to adequately describe. Herman (1992) proposed the term "disorder of extreme stress" to capture the complexity of these clinical presentations, which included profound alterations in identity, relationships, and emotional regulation.


C-PTSD is today recognized as a diagnostic category in the ICD-11 of the World Health Organization (WHO, 2019), which distinguishes it from PTSD by the presence of three additional clusters beyond the basic traumatic symptoms: affective dysregulation, alterations of identity, and difficulties in interpersonal relationships. It is not, however, present in the DSM-5-TR (APA, 2022), and the scientific debate on this distinction remains open.


The crucial difference does not concern only the number of symptoms, but their quality and depth. In C-PTSD, trauma does not exclusively affect memories of the event: it reshapes the way the person sees themselves, relates to others, and regulates their inner world. As Courtois and Ford (2009) write, complex trauma leaves imprints that cut across identity, memory, the body, and relationships — making the self itself the terrain upon which trauma continues to act.


Neurobiology of complex trauma: the body remembers

One of the most powerful insights of contemporary trauma research is that the body does not forget. Van der Kolk (2014) documented how trauma is stored not only in explicit memories — that is, in the narratives we can tell — but in somatic patterns, autonomic responses, and physical sensations that activate automatically even in the absence of conscious recollection.


This is particularly relevant in C-PTSD. In those who have experienced chronic and early trauma, the autonomic nervous system may remain stably altered, oscillating between states of hyperactivation (anxiety, hypervigilance, aggression) and states of hypoactivation (dissociation, numbness, emotional detachment). Porges (2011), with his polyvagal theory, provided a neurobiological model for understanding these oscillations: the nervous system, faced with a threat perceived as inescapable, activates immobilization responses that over time become chronic states difficult to modulate.


The "window of tolerance" — a concept developed by Siegel (1999) — describes that optimal zone of activation in which a person can process experiences without being overwhelmed or dissociated. In survivors of complex trauma, this window tends to be significantly narrowed: even low-intensity stimuli can trigger disproportionate alarm reactions, or conversely, a sudden emotional withdrawal. Therapeutic work aims, among other things, to progressively widen this window.


Relational trauma and attachment theory

One of the most distinctive aspects of C-PTSD is its relational origin. In many cases, complex trauma develops in contexts where the source of danger coincides with the caregiving figure: an abusive parent, a violent partner, a chronic family environment of maltreatment. Bowlby (1988) had already intuited how early attachment experiences shaped "internal working models" — mental representations of oneself, others, and relationships — that tend to persist into adulthood and organize the way we interpret relational experiences.


When the caregiver is simultaneously a source of danger and of nurturing, the child finds themselves in an irresolvable paradox: the attachment system and the defense system are activated simultaneously by the same stimulus. Main and Hesse (1990) described this pattern as "disorganized attachment," which is associated with significant negative outcomes in terms of emotional regulation, identity, and long-term mental health.


In C-PTSD, this relational background translates into dysfunctional interpersonal schemas: difficulty trusting, a tendency to re-enact victimization or control dynamics in adult relationships, oscillations between idealization and devaluation. It is precisely this relational pervasiveness that makes C-PTSD clinically overlapping with Borderline Personality Disorder (BPD), with which it shares numerous symptoms — so much so that some authors have proposed considering BPD as a severe form of complex trauma (Herman, 1992; van der Kolk, 2014).


The diagnostic challenge: recognizing without confusing

One of the most significant challenges in clinical practice is that C-PTSD is often mistaken for other disorders. Its symptoms overlap with those of major depression, Borderline Personality Disorder, dissociative disorders, and bipolar disorder. This overlap is not accidental: many of these disorders can be read as adaptive responses to experiences of chronic trauma (Felitti et al., 1998).


The ACE (Adverse Childhood Experiences) study, conducted by Felitti and collaborators (1998) on over 17,000 adults, definitively documented the dose-response relationship between early adverse experiences and negative outcomes on physical and mental health over the course of a lifetime. More adverse experiences, the higher the probability of developing mood disorders, addictions, cardiovascular disease, and complex psychological disturbances. This study revolutionized the clinical perspective, shifting the question from "what is wrong with you?" to "what happened to you?"


Accurate clinical assessment therefore remains indispensable. Specific instruments such as the International Trauma Questionnaire (ITQ; Cloitre et al., 2018) have been developed precisely to discriminate between PTSD and C-PTSD according to ICD-11 criteria, offering the clinician an empirically validated tool in the diagnostic formulation.


Treatments: a tailored approach

The treatment of trauma — both PTSD and C-PTSD — today draws on evidence-based therapeutic approaches of proven efficacy. Among the most widely used:

  • EMDR (Eye Movement Desensitization and Reprocessing) is recognized by the WHO as a first-line treatment for PTSD. The protocol developed by Shapiro (2018) acts on unprocessed traumatic memories, facilitating their integration into autobiographical memory through bilateral stimulation. In cases of complex trauma, the protocol must be adapted with more extensive preparatory phases and careful attention to emotional stabilization.

  • Trauma-Focused Cognitive Behavioral Therapy (TF-CBT) has a solid evidence base for PTSD, acting on dysfunctional thoughts, emotions, and avoidance behaviors connected to the traumatic experience (Cohen et al., 2006). In C-PTSD it is often integrated with emotion regulation interventions.

  • Sensorimotor Psychotherapy, developed by Ogden et al. (2006), works explicitly on the body's responses to trauma, integrating somatic awareness and body-oriented interventions. This approach is particularly relevant in C-PTSD, where traumatic traces are often stored in the body rather than in explicit narrative.

  • Trauma-oriented psychodynamic therapies explore the meaning of traumatic experiences and their impact on relationships and identity. They work in depth on internal working models and on the transferential dynamics that trauma activates within the therapeutic context.


In general, treatment of C-PTSD follows a phase-based model — stabilization, trauma processing, integration — as proposed by Herman (1992) and subsequently taken up by numerous authors. The therapeutic relationship itself becomes a fundamental instrument: it is in the encounter with a trustworthy other that the attachment system can slowly reorganize itself.

Conclusions

Distinguishing PTSD from C-PTSD does not simply mean applying different diagnostic categories. It means recognizing that people who have experienced chronic, relational, and early trauma carry with them a suffering that has reshaped not only their memories, but their identity, their body, and their way of being in the world. Acknowledging this complexity is the first step toward offering care that is truly equal to the experience lived.


As Herman (1992) wrote, recovery from trauma is fundamentally an act of reclaiming oneself: it is not about forgetting, but about integrating — restoring the past to its place in time, and restoring the present to its possibility of being different.


References

American Psychiatric Association. (2022). Diagnostic and statistical manual of mental disorders (5th ed., text rev.). https://doi.org/10.1176/appi.books.9780890425787


Bowlby, J. (1988). A secure base: Parent-child attachment and healthy human development. Basic Books.


Bremner, J. D. (2006). Traumatic stress: Effects on the brain. Dialogues in Clinical Neuroscience, 8(4), 445–461. https://doi.org/10.31887/DCNS.2006.8.4/jbremner


Cloitre, M., Shevlin, M., Brewin, C. R., Bisson, J. I., Roberts, N. P., Maercker, A., Karatzias, T., & Hyland, P. (2018). The International Trauma Questionnaire: Development of a self-report measure of ICD-11 PTSD and complex PTSD. Acta Psychiatrica Scandinavica, 138(6), 536–546. https://doi.org/10.1111/acps.12956


Cohen, J. A., Mannarino, A. P., & Deblinger, E. (2006). Treating trauma and traumatic grief in children and adolescents. Guilford Press.


Courtois, C. A., & Ford, J. D. (2009). Treating complex traumatic stress disorders: An evidence-based guide. Guilford Press.


Felitti, V. J., Anda, R. F., Nordenberg, D., Williamson, D. F., Spitz, A. M., Edwards, V., Koss, M. P., & Marks, J. S. (1998). Relationship of childhood abuse and household dysfunction to many of the leading causes of death in adults. American Journal of Preventive Medicine, 14(4), 245–258. https://doi.org/10.1016/S0749-3797(98)00017-8


Herman, J. L. (1992). Trauma and recovery: The aftermath of violence — from domestic abuse to political terror. Basic Books.


Main, M., & Hesse, E. (1990). Parents' unresolved traumatic experiences are related to infant disorganized attachment status. In M. T. Greenberg, D. Cicchetti, & E. M. Cummings (Eds.), Attachment in the preschool years (pp. 161–182). University of Chicago Press.


Ogden, P., Minton, K., & Pain, C. (2006). Trauma and the body: A sensorimotor approach to psychotherapy. W. W. Norton & Company.


Porges, S. W. (2011). The polyvagal theory: Neurophysiological foundations of emotions, attachment, communication, and self-regulation. W. W. Norton & Company.


Shapiro, F. (2018). Eye movement desensitization and reprocessing (EMDR) therapy: Basic principles, protocols, and procedures (3rd ed.). Guilford Press.


Siegel, D. J. (1999). The developing mind: How relationships and the brain interact to shape who we are. Guilford Press.


van der Kolk, B. A. (2014). The body keeps the score: Brain, mind, and body in the healing of trauma. Viking.


World Health Organization. (2019). International classification of diseases for mortality and morbidity statistics (11th rev.). https://icd.who.int/


Comments


© 2035 by Charley Knox. Powered and secured by Wix

bottom of page